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  •   University of Thessaly Institutional Repository
  • Επιστημονικές Δημοσιεύσεις Μελών ΠΘ (ΕΔΠΘ)
  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ.
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  •   University of Thessaly Institutional Repository
  • Επιστημονικές Δημοσιεύσεις Μελών ΠΘ (ΕΔΠΘ)
  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ.
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Complexity of persistent type II endoleak associated with sac expansion after endovascular abdominal aortic aneurysm repair

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Author
Spanos K., Rountas C., Giannoukas A.D.
Date
2015
Language
en
DOI
10.1177/1708538114562021
Keyword
aged
Article
case report
colon tumor
computed tomographic angiography
computer assisted tomography
endoleak
endovascular aneurysm repair
follow up
human
inferior mesenteric artery
laparotomy
lung embolism
magnetic resonance angiography
male
priority journal
sigmoid carcinoma
adverse effects
Aortic Aneurysm, Abdominal
aortography
artificial embolization
blood vessel prosthesis
blood vessel transplantation
devices
endoleak
endovascular surgery
multimodal imaging
procedures
reoperation
stent
time factor
treatment outcome
Aged
Aortic Aneurysm, Abdominal
Aortography
Blood Vessel Prosthesis
Blood Vessel Prosthesis Implantation
Embolization, Therapeutic
Endoleak
Endovascular Procedures
Humans
Male
Multimodal Imaging
Reoperation
Stents
Time Factors
Tomography, X-Ray Computed
Treatment Outcome
SAGE Publications Ltd
Metadata display
Abstract
Type II endoleak after endovascular aortic aneurysm repair still remains the Achilles’ heel of the treatment, the source of which regularly is difficult to identify and treat. We present a patient with a persistent type II endoleak associated with a continuous aneurysm sac expansion after endovascular aortic aneurysm repair for which many diagnostic modalities were used during his follow-up such as duplex scan, computed tomography angiography and magnetic resonance angiography. Attempts were undertaken to treat the source of endoleak including coil micro-embolisation of lumbar arteries and subsequent open ligation of the inferior mesenteric artery, but they failed to eliminate the endoleak. Finally, a middle sacral artery was identified as the source of the endoleak. At that time, the patient was subjected to surgery for sigmoid carcinoma, and simultaneously, a ligation of the sacral artery was undertaken which eventually eliminated the endoleak completely. This case highlights that type II endoleak may be evoked by various sources and there can be a great difficulty to identify these feeding vessels; thus, careful planning for its management is mandatory. © 2014, © The Author(s) 2014.
URI
http://hdl.handle.net/11615/79296
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  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ. [19735]
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