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  •   Ιδρυματικό Αποθετήριο Πανεπιστημίου Θεσσαλίας
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  •   Ιδρυματικό Αποθετήριο Πανεπιστημίου Θεσσαλίας
  • Επιστημονικές Δημοσιεύσεις Μελών ΠΘ (ΕΔΠΘ)
  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ.
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Ιδρυματικό Αποθετήριο Πανεπιστημίου Θεσσαλίας
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Regulatory B cells: New players in inflammatory and autoimmune rheumatic diseases

Thumbnail
Συγγραφέας
Sakkas L.I., Daoussis D., Mavropoulos A., Liossis S.-N., Bogdanos D.P.
Ημερομηνία
2019
Γλώσσα
en
DOI
10.1016/j.semarthrit.2018.10.007
Λέξη-κλειδί
autoantibody
interleukin 10
anaphylactoid purpura
ANCA associated vasculitis
ankylosing spondylitis
autoimmune disease
disease association
ex vivo study
giant cell arteritis
human
inflammatory bowel disease
inflammatory disease
lung fibrosis
nonhuman
pathogenesis
priority journal
psoriatic arthritis
reactive arthritis
regulatory B lymphocyte
Review
rheumatic disease
rheumatoid arthritis
Sjoegren syndrome
spondylarthritis
systematic review
systemic lupus erythematosus
systemic sclerosis
Th1 cell
Th17 cell
autoimmune disease
immunology
inflammation
regulatory B lymphocyte
rheumatic disease
Autoimmune Diseases
B-Lymphocytes, Regulatory
Humans
Inflammation
Rheumatic Diseases
W.B. Saunders
Εμφάνιση Μεταδεδομένων
Επιτομή
Objective: Regulatory B cells (Bregs) are a new subset of B cells with immunoregulatory functions, mainly through IL-10 production. Bregs suppress inflammatory Th1 and Th17 differentiation and induce Tregs suppressing autoimmune diseases. The aim of the study was to review the literature related to Bregs in autoimmune rheumatic diseases (ARDs). Methods: A literature review of publications in PUBMED published in English was performed using the relevant combinations of terms. Results: All relevant publications are discussed. Overall, recent studies in rheumatic diseases found Bregs to be decreased in ANCA-associated vasculitides (AAV) and in systemic sclerosis (SSc), particularly in SSc-associated lung fibrosis. In AAV Bregs levels are negatively correlated with autoantibody levels whereas in SSc this association is less clear but there is an inverse association with Th1 and Th17 cells. In rheumatoid arthritis (RA), Bregs were decreased, particularly in RA-associated lung fibrosis. In psoriatic arthritis IL-10 + Bregs are decreased and inversely associated with Th1 and Th17 cells. In systemic lupus erythematosus (SLE), the role of Bregs is unclear. In experimental diseases, when Bregs were expanded ex-vivo, they ameliorated established disease. Conclusion: Bregs appear to be a new player in the pathogenesis of ARDs, and may offer a new strategy for therapeutic intervention. © 2018 Elsevier Inc.
URI
http://hdl.handle.net/11615/78727
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  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ. [19735]

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    SARS-CoV-2 pathophysiology and its clinical implications: An integrative overview of the pharmacotherapeutic management of COVID-19 

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    Common manifestations of COVID-19 are respiratory and can extend from mild symptoms to severe acute respiratory distress. The severity of the illness can also extend from mild disease to life-threatening acute respiratory ...
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