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  • Επιστημονικές Δημοσιεύσεις Μελών ΠΘ (ΕΔΠΘ)
  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ.
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  • Επιστημονικές Δημοσιεύσεις Μελών ΠΘ (ΕΔΠΘ)
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HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia

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Auteur
de Barrios O., Galaras A., Trincado J.L., Azagra A., Collazo O., Meler A., Agraz-Doblas A., Bueno C., Ballerini P., Cazzaniga G., Stam R.W., Varela I., De Lorenzo P., Valsecchi M.G., Hatzis P., Menéndez P., Parra M.
Date
2021
Language
en
DOI
10.1038/s41375-020-01097-x
Sujet
doxycycline
histone deacetylase 7
HDAC7 protein, human
histone deacetylase
mixed lineage leukemia protein
acute lymphoblastic leukemia
Article
B lymphocyte differentiation
bioinformatics
cancer prognosis
cancer survival
CCL5 gene
CD86 gene
chromosome translocation 11
chromosome translocation 4
clinical article
clinical outcome
cohort analysis
controlled study
disease association
gene
gene expression
gene expression level
gene ontology
gene overexpression
genetic analysis
HDAC7 gene
human
human cell
infant
LRP1 gene
MLL gene
pathogenesis
phenotype
pre B lymphocyte
proB acute lymphoblastic leukemia
protein function
reverse transcription polymerase chain reaction
RNA sequence
SEMK2 cell line
transcriptomics
VEGFA gene
acute lymphoblastic leukemia
gene translocation
genetics
Histone Deacetylases
Humans
Infant
Myeloid-Lymphoid Leukemia Protein
Precursor Cell Lymphoblastic Leukemia-Lymphoma
Translocation, Genetic
Springer Nature
Afficher la notice complète
Résumé
[No abstract available]
URI
http://hdl.handle.net/11615/73126
Collections
  • Δημοσιεύσεις σε περιοδικά, συνέδρια, κεφάλαια βιβλίων κλπ. [19735]

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