Loss of C9orf72 function leads to autoimmunity

dc.creator	Sakkas L.I., Bogdanos D.P., Kousvelari E.E.	en
dc.date.accessioned	2023-01-31T09:53:18Z
dc.date.available	2023-01-31T09:53:18Z
dc.date.issued	2017
dc.identifier	10.21037/atm.2017.01.33
dc.identifier.issn	23055839
dc.identifier.uri	http://hdl.handle.net/11615/78725
dc.description.abstract	[No abstract available]	en
dc.language.iso	en	en
dc.source	Annals of Translational Medicine	en
dc.source.uri	https://www.scopus.com/inward/record.uri?eid=2-s2.0-85013141361&doi=10.21037%2fatm.2017.01.33&partnerID=40&md5=fc801c0ab10fd0c78d71118f67571f53
dc.subject	C9orf72 protein	en
dc.subject	HLA antigen class 2	en
dc.subject	inflammasome	en
dc.subject	regulator protein	en
dc.subject	unclassified drug	en
dc.subject	Article	en
dc.subject	autoimmune disease	en
dc.subject	autoimmunity	en
dc.subject	autophagy	en
dc.subject	B lymphocyte	en
dc.subject	bone marrow cell	en
dc.subject	C9orf72 gene	en
dc.subject	COPA gene	en
dc.subject	endoplasmic reticulum stress	en
dc.subject	environmental factor	en
dc.subject	gene	en
dc.subject	gene function	en
dc.subject	human	en
dc.subject	loss of function mutation	en
dc.subject	microglia	en
dc.subject	nonhuman	en
dc.subject	pathogenesis	en
dc.subject	T lymphocyte	en
dc.subject	AME Publishing Company	en
dc.title	Loss of C9orf72 function leads to autoimmunity	en
dc.type	journalArticle	en

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