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dc.creatorNikolaidis, M. G.en
dc.creatorKyparos, A.en
dc.creatorDipla, K.en
dc.creatorZafeiridis, A.en
dc.creatorSambanis, M.en
dc.creatorGrivas, G. V.en
dc.creatorPaschalis, V.en
dc.creatorTheodorou, A. A.en
dc.creatorPapadopoulos, S.en
dc.creatorSpanou, C.en
dc.creatorVrabas, I. S.en
dc.date.accessioned2015-11-23T10:41:03Z
dc.date.available2015-11-23T10:41:03Z
dc.date.issued2012
dc.identifier10.3109/1354750x.2011.635805
dc.identifier.issn1354-750X
dc.identifier.urihttp://hdl.handle.net/11615/31370
dc.description.abstractTwenty males ran either on a level treadmill (nonmuscle-damaging condition) or on a downhill treadmill (muscle-damaging condition). Blood and urine samples were collected before and after exercise (immediately after, 1h, 4h, 24h, 48h, and 96h). The following assays were performed: F-2-isoprostanes in urine, protein carbonyls in plasma, glutathione, superoxide dismutase, glutathione peroxidase, and catalase in erythrocytes. The main finding was that monophasic redox responses were detected after nonmuscle-damaging exercise compared to the biphasic responses detected after muscle-damaging exercise. Based on these findings, muscle-damaging exercise may be a more appropriate experimental model to induce physiological oxidative stress.en
dc.source.uri<Go to ISI>://WOS:000299750500005
dc.subjectEccentric exerciseen
dc.subjectmuscle damageen
dc.subjectreactive oxygen speciesen
dc.subjectisoprostanesen
dc.subjectoxidative stressen
dc.subjectbiomarkersen
dc.subjectINDUCED OXIDATIVE STRESSen
dc.subjectANTIOXIDANT SUPPLEMENTATIONen
dc.subjectLIPID-PEROXIDATIONen
dc.subjectAEROBIC EXERCISEen
dc.subjectSKELETAL-MUSCLEen
dc.subjectHUMAN PLASMAen
dc.subjectVITAMIN-Een
dc.subjectQUANTIFICATIONen
dc.subjectGENERATIONen
dc.subjectRUNNERSen
dc.subjectBiotechnology & Applied Microbiologyen
dc.subjectToxicologyen
dc.titleExercise as a model to study redox homeostasis in blood: the effect of protocol and sampling pointen
dc.typejournalArticleen


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